Methyltestosterone is an orally available form of the primary male
androgen testosterone. Looking at the structure of this steroid, we see it
is basically just testosterone with an added methyl group at the c-17
alpha position (a c-17 alpha alkylated substance). Alkylation such as this
is necessary when administering testosterone (and other steroids) orally,
as without it the liver will destroy most of the steroid during the "first
pass". The resultant compound "methylated-testosterone" was among the
first functional oral steroids to be produced. This field of research has
consequently improved greatly over the years, and today methyltestosterone
is quite crude in comparison to many of the other orals that were
subsequently developed.
The action
of this steroid is somewhat androgenic, with a moderate anabolic effect.
As is typically seen with 17 alpha methylation, the resulting steroid has
lower anabolic activity than its parent testosterone. Additionally it is
extremely estrogenic, another property that seems to be enhanced when this
alteration is present (when the steroid is receptive to the aromatase
enzyme). The problem seems to be its conversion to the more biologically
active estrogen 17-alpha methylestradiol. 17-alpha methylation in fact
slows aromatization, however the potent nature of 1 -methylestradiol more
than compensates for this. Additionally it has a very short half-life in
the body, so the drug needs to be administered several times daily if a
consistent blood level is to be obtained. All of this heightens the ratio
of side effects to muscle growth enough to make methyltestosterone a very
inefficient muscle-building drug. In order to administer an effective
amount of hormone, the user simply must deal with too many estrogenic side
effects, including water retention and gynecomastia, which can be very
troublesome with this steroid. One may choose to addition an anti-estrogen
such as Nolvadex and/or
Proviron to combat related side effects, which should effectively
minimize their intensity enough to make a cycle tolerable. The powerful
antiaromatase Arimidex is a notably more
effective option when dealing with aromatizable steroids, as it shows
great ability to stop the conversion of androgens to estrogens.
Just as we
see with its parent testosterone, methyltestosterone has a high rate of
conversion to DHT (in this case 17alpha-methyldihydrotestosterone). This
metabolite is of course more active than methyltestosterone, and likewise
responsible for many of the unwanted androgenic side effects encountered
with use. Oily skin, acne, body/facial hair growth and hair loss are
likewise all common with this steroid, and may present themselves very
early into a cycle. Also seen with use of this compound is an increased
level of aggression, an effect commonly associated with testosterone and
other strong androgens. The addition of Proscar
could quite prove useful with this steroid, inhibiting the conversion of
methyltestosterone into methyl-DHT in many target tissues and lowering the
impact of related side effects. Again, this ancillary drug is in most
instances considerably more expensive than the steroid it is being used to
treat.
A strong
androgen such as methyltest obviously has little to offer female athletes
except virilization symptoms. In this arena, methyltestosterone should be
strictly avoided. The only time females should really be taking this
potent steroid is when indicated for a specific medical application. While
not a new concept, using an androgen to treat the symptoms of menopause
has been catching on in recent years nonetheless.
Being a c-17
alpha alkylated compound, methyltestosterone also places notable stress on
the liver. This is further amplified when looking at the amount of drug
necessary to see an anabolic effect. Those willing to use this drug for
actual muscle growth ordinarily find that a daily dosage of 40-50 mg is
necessary (at a minimum) for acceptable results. This is quite a lot of
c-17aa steroid for the liver to process, especially when comparing it to
the effect seen with a much smaller amount of
Dianabol or Winstrol. One should therefore
limit a cycle of this drug to no more than 6 or 8 weeks, after which a
longer break should be taken from all "toxic" oral steroids. One should
also be prepared for a substantial loss of mass and bodyweight at the
conclusion of each cycle with methyltest. This is due to a combination
retained water being excreted, and the suppression of endogenous
testosterone production during intake. A testosterone stimulating drug
such as Clomid/Nolvadex
and/or HCG is therefore used to restore hormonal
balance.
Clearly
methyltestosterone is not a very advanced compound. While it is close
derivative of testosterone, with potential as such, it offers us little
benefit in practice. The short activity and high rate of estrogenic
activity generally make this product too troublesome to use for
performance enhancement. The only commonly accepted application for
methyltestosterone is to stimulate aggression in the user. Powerlifters,
bodybuilders and competitive athletes often attempt to "harness" this
aggression, looking for extra intensity in a training session or
competition. Additionally, many methyltest tabs are designed for
sublingual administration, or to be placed under the tongue and left to
dissolve. These tabs can generally be identified by a pleasant tasting
citrus flavor, which is most often included. Sublingual intake is an added
benefit for aggression stimulating purposes, providing fast (albeit
incomplete) absorption of the drug. A couple of tablets placed under the
tongue before a visit to the gym can make for an intense, and possibly
more productive, workout session. Aside from this methyltest offers little
except side effects. It is quite toxic, elevating liver enzymes and
causing acne, gynecomastia, aggression and water retention quite easily.
Were one to tolerate these side effects, methyltest will offer little more
than poor quality (but bulky) gains. One looking for quality muscle
building steroid should likewise look elsewhere.
::
Buy
Methyltestosterone
::
